Annals of Indian Academy of Neurology
EDITORIAL
Year
: 2007  |  Volume : 10  |  Issue : 4  |  Page : 197-

From muscular dystrophy to chickenpox


Sanjeev V Thomas 
 Department of Neurology, SCTIMST, Trivandrum, India

Correspondence Address:
Sanjeev V Thomas
Department of Neurology, SCTIMST, Trivandrum
India




How to cite this article:
Thomas SV. From muscular dystrophy to chickenpox.Ann Indian Acad Neurol 2007;10:197-197


How to cite this URL:
Thomas SV. From muscular dystrophy to chickenpox. Ann Indian Acad Neurol [serial online] 2007 [cited 2020 Oct 24 ];10:197-197
Available from: https://www.annalsofian.org/text.asp?2007/10/4/197/37811


Full Text

The fifteenth annual meeting of the IAN in 2007 in Mumbai was a grand success. Most of the presentations and lectures were of high standards. A large contingent of delegates from the British Neurological Association enriched this meeting. At present, this journal has a stronger financial position; thanks to the generous gift from the organizers of the 2007 annual meeting in Mumbai and the enhanced annual grant from the IAN. We hope that the readers will benefit from these encouraging gestures.

This issue of the AIAN has a new column - "Statistically speaking." We propose to bring out a series of articles on frequently used and often less understood statistical tests and procedures. Prof. Kameshwar Prasad has kindly agreed to present this column for the journal. We hope that this column would enable the readers to sharpen their statistical skills and make better interpretation of the statistically heavy weight articles.

Limb girdle muscular dystrophy (LGMD) is a clinical syndrome that causes severe disability to approximately three million individuals in India. The precise diagnosis of LGMD requires concerted effort of clinicians, neuropathologists, immunohistochemists and molecular geneticists. Such comprehensive facility is available only in a few centers in India. A detailed review of the neuropathology of LGMD by a subject expert is included in this issue. We hope that in the near future the neurologists in India who have special interest in neuromuscular disorders would join hands together to develop a national registry of LGMD.

Chickenpox is an infection caused by Varicella Zoster Virus (VZV). This virus tends to infect a wide range of cell types in the central and peripheral nervous system, thereby causing a variety of clinical syndromes. The neurological complications include myelitis, menigoencephalitis, large-vessel granulomatous arteritis that lead to stroke, small vessel encephalitis, ventriculitis, Guillain Barre Syndrome and postherpetic neuralgia. These complications are difficult to diagnose when the infection occurs without skin manifestations. The infection caused by VZV in an immunocompromized host can be more serious. Epidemiological studies indicate that people living in India and other tropical countries get sero-converted for VZV at a later age than those living in temperate regions. Therefore, a greater number of adults are susceptible to VZV infection in these countries in comparison to other countries in the temperate region. The morbidity and mortality from chicken pox is more when the disease occurs in older persons. Besides, the infection during pregnancy can lead to congenital varicella infection in the offspring. There have been very few publications from India with regard to this important disease. We have presented one article that provides details of the neurological complications of chickenpox in Kerala, India.

Aneurysmal subarachnoid hemorrhage (SAH) is an important cause of mortality in stroke. Noninvasive imaging of the aneurysms with high-resolution CT angiography and MR angiography have eased some of the difficulties to diagnose and monitor cerebral aneurysms. Several interventional radiological procedures have recently become available in addition to the well-established surgical procedures to treat cerebral aneurysms. Approximately 10-19% of the aneurysms in the anterior cerebral circulation tend to rebleed after an initial bleed. Mortality due to SAH in the second week after the rupture of an intracranial aneurysm is largely due to the rebleed. One of the articles in this issue discusses the potential factors that may influence rebleed in persons with aneurysmal SAH.

Let me conclude this editorial with season's greetings to all of the readers.