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Figure 1: (a and b) Brain magnetic resonance imaging (MRI) of the patient: (a) the T2-weighted image demonstrated bilateral hippocampal hyperintensity, (b) the MRI fluid-attenuated inversion recovery image showed bilateral hippocampal hyperintensity (arrows). (c–k) Positron emission tomography based on 2-deoxy-2-[18F] fluoro-D-glucose showed a diffuse decrease in glucose metabolism (green color) in the cerebral cortex and cerebellar hemisphere, but increased metabolism (yellower/redder color) in the bilateral caudate putamen, lenticular nucleus, and dorsal thalamus. Increased glucose metabolism was also observed in the right hippocampus

Figure 1: (a and b) Brain magnetic resonance imaging (MRI) of the patient: (a) the T2-weighted image demonstrated bilateral hippocampal hyperintensity, (b) the MRI fluid-attenuated inversion recovery image showed bilateral hippocampal hyperintensity (arrows). (c–k) Positron emission tomography based on 2-deoxy-2-[<sup>18</sup>F] fluoro-D-glucose showed a diffuse decrease in glucose metabolism (green color) in the cerebral cortex and cerebellar hemisphere, but increased metabolism (yellower/redder color) in the bilateral caudate putamen, lenticular nucleus, and dorsal thalamus. Increased glucose metabolism was also observed in the right hippocampus